Paul Saladino· MD
this is probably the fatty acid that our lipid containing cells in our adipose tissue in our bellies are releasing to signal to the rest of the body to remain insulin sensitive
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The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
this is probably the fatty acid that our lipid containing cells in our adipose tissue in our bellies are releasing to signal to the rest of the body to remain insulin sensitive
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when we increase the amount of fat in our diet and when we decrease the amount of carbohydrates there are different signals from the adipose tissue because of declining levels of insulin right so what we know is that if we don't eat carbohydrates if we eat a high if we eat a high fat low carb diet with a moderate amount of protein insulin signal drops and when the insulin signal at the adipocytes drops they change the fatty acids that they put out into the blood and it shifts from correct meaning if i'm wrong here palmitoleic acid to palmitate and then palmitate circulates to the muscle cells and kind of gives them the signal hey refuse glucose we've got plenty of free fatty acids spare the glucose for the brain and the red blood cells