Controversial Thoughts: How to get Omega-3 without Rancid Fish Oil

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Paul Saladino27m · 2020-09-03

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43

Claims Supported

35

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35 of 43 claims supported

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35

Well SupportedExcessive lipolysis and broken fat cells, potentially caused by polyunsaturated fatty acid breakdown products like linoleic acid, can lead to insulin resistance.Well SupportedChickens and pigs are typically fed diets higher in linoleic acid than they would consume in the wild.Well SupportedMost individuals can obtain sufficient omega-3 fatty acids from grass-fed animal fats like suet or tallow, negating the need for excessive omega-3 consumption.Well SupportedEPA and DHA can be obtained from egg yolks, and ALA can be converted to DHA when linoleic acid from seed oils is not in excess.Well Supported

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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.

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Excess omega-6s can inhibit the conversion of alpha-linolenic acid to EPA and DHA.

Well SupportedIf omega-6 fatty acids are low in the diet, the body can convert alpha-linolenic acid (ALA) found in animal fats into EPA, DPA, and DHA.

Well SupportedFeeding diabetic animals a high polyunsaturated to saturated fat diet increases membrane linoleic acid content and is associated with increased insulin binding in non-diabetic animals.

Well SupportedGrass-fed steaks have higher levels of omega-3 fatty acids, vitamin K2, and conjugated linoleic acid compared to grain-fed steaks.

Well SupportedIn a six-month trial, diets with higher fat and lower carbohydrate content resulted in less weight loss than diets with lower fat and higher carbohydrate content in metabolically healthy adults, despite being isocaloric.

Not SupportedDiets higher in soybean oil were associated with less weight loss in a hypocaloric study.

Partially SupportedAn animal-based diet including meat and organs, and potentially desiccated organ supplements, has been helpful for individuals with metabolic issues and allergies.

Partially SupportedIncreased consumption of vegetable oils since 1960 correlates with higher rates of diabetes, obesity, and chronic disease.

Partially SupportedDietary stearic acid reduces visceral adipose tissue.

Partially SupportedStearic acid, prevalent in animal fat, suet, and cacao butter, may have an anti-metastatic effect in breast cancer.

Partially SupportedShrinking adipocytes can be achieved with saturated fats like stearic acid found in animal fats.

Partially SupportedAn adipocyte-centered model of metabolic dysfunction proposes that insulin-resistant adipocytes are desirable because they do not grow and instead release stored fats for the body to burn, promoting satiety.

Partially SupportedEvolutionarily, stearic acid is predominantly found in animal foods, particularly suet.

Partially SupportedRuminants can convert linoleic acid to ruminic acid and produce stearic acid in their fat.

Partially SupportedIncreased insulin binding to fat cells, driven by high PUFA diets, leads to growing fat cells and can result in metabolic dysfunction.

Limited ResearchCarbohydrates may not be the primary drivers of metabolic dysfunction in the US.

Partially SupportedLow linoleic acid intake and high stearic acid intake can improve weight loss stalls.

Not SupportedFlax seeds are not a good source of omega-3s (EPA and DHA) and may cause gastrointestinal side effects.

Partially SupportedWarfarin inhibits vitamin K formation, which may explain observed correlations between warfarin use and cardiovascular disease.

Not SupportedPolyunsaturated fats affect adipocytes, making them grow and become more insulin sensitive, which is considered a negative physiological outcome.

Partially SupportedA diet rich in CLA and butter, supplemented at super-physiological amounts, increases lipid peroxidation without affecting atherosclerotic, inflammatory, or diabetic risk markers in young healthy men.

Partially SupportedIn Japan from 1950-2010, decreased total energy and carbohydrate intake coincided with increased lipid energy intake and a sharp rise in diabetes prevalence.

Not SupportedSupplementation with conjugated linoleic acid (CLA) is not recommended; consuming nose-to-tail from ancestral diets is preferred.

Not SupportedStatins may have negative effects beyond their potential cleotropic benefits.

Partially SupportedFeeding animals a diet high in polyunsaturated fats (PUFA) relative to saturated fats (SFA) increases membrane linoleic acid content and prevents the decrease of arachidonic acid in membrane phospholipids.

Partially SupportedExcessive supplementation with conjugated linoleic acid (CLA) can lead to oxidation because it is a polyunsaturated fatty acid.

Partially SupportedHigh levels of linoleic acid in red blood cells are associated with insulin resistance and metabolic unhealth.

Partially SupportedConjugated linoleic acid (CLA) is present in very low amounts in red meat.

Partially SupportedAdipocytes should be insulin resistant, which is achieved by eating like one's ancestors.

Partially SupportedAchieving low linoleic acid intake and high stearic acid intake, by consuming nose-to-tail with organs and suet, allows the body to convert ALA to EPA and DHA ancestrally.

Not SupportedHigh intake of polyunsaturated fats causes adipocytes to grow by increasing insulin sensitivity.